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Dumping syndrome NCLEX practice question "select all that apply". This NCLEX review question will test your nursing knowledge on how to educate a patient about decreasing signs and symptoms experienced in dumping syndrome.
What is dumping syndrome? It is a condition where food from the stomach enters too rapidly into the small intestine.
Signs and symptoms of dumping syndrome can present 30 minutes to 3 hours AFTER eating a meal and can include: abdominal cramping, nausea, hypotension, tachycardia, bloating, diaphoresis, weakness, drop in blood glucose etc.
Patients who've had stomach surgery such as gastric resection or similar are at risk for dumping syndrome.
This video is part of a weekly NCLEX review series where I will be going over NCLEX style questions with you. I will be helping you analyze each question, and walk you through how to select the correct option.
NCLEX questions require critical thinking and you must know how to use your nursing knowledge to gather the facts and analyze what the question is asking.
This specific question is a type of question that wants to know based on your morning assessment findings which finding is abnormal and requires immediate nursing action.
NCLEX Question:
A patient is post-op from a gastric resection for treatment of peptic ulcer disease. One hour after eating meals, the patient exhibits diaphoresis, tachycardia, and hypotension. In addition, the patient reports feeling abdominal cramps, weakness, and nausea. Which options below can be incorporated in the patient's plan of care to help alleviate the patient's signs and symptoms? Select all that apply:
A. Wait 30 minutes after meals to consume liquids.
B. Sit up for 30 minutes after eating.
C. Consume high amounts of dairy products daily.
D. Eat 5-6 small meals a day rather than 3 large meals.
E. When symptoms present, eat cold or hot food to help decrease symptoms.
Watch the video for the correct answers.
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Title: Familial adenomatous polyposis
Familial adenomatous polyposis: A genetic disease characterized by the presence of numerous precancerous polyps in the colon and rectum. The polyps usually begin to form at puberty, and colon cancer almost always develops later in life. Abbreviated FAP. FAP is inherited as an autosomal dominant trait. Most people who receive the gene manifest the disease, although the expression of FAP can vary markedly from person to person. The gene that is mutated in FAP is the APC (adenomatous polyposis coli) gene on chromosome 5. Surgery is often necessary to remove the colon in order to prevent the development of cancer. A milder type of familial adenomatous polyposis has been identified that is inherited in an autosomal recessive manner. This is referred to as autosomal recessive familial adenomatous polyposis and is caused by mutations in a different gene known as MUTYH. Also known as familial polyposis, familial polyposis coli.
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In the 1980s, HIV began spreading throughout North America and the world at large. From "gay cancer" to the myth of Patient Zero, these are some of the stories that emerged from those first few years.
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Source/Further reading:
The Basics of HIV and AIDS:
https://www.hiv.gov/hiv-basics
https://www.ncbi.nlm.nih.gov/p....mc/articles/PMC32344
CDC Opportunistic Infections:
https://www.cdc.gov/hiv/basics..../livingwithhiv/oppor
CDC Incidence rates by population sub-groups:
https://www.cdc.gov/hiv/basics/statistics.html
https://www.census.gov/quickfa....cts/fact/table/US/PS
AIDS/HIV origins and history:
https://www.healthline.com/health/hiv-aids/history
https://www.hiv.gov/hiv-basics/overview/history/hiv-and-aids-timeline
https://jech.bmj.com/content/67/6/473
https://www.nature.com/articles/nature19827
https://hivhistory.org/panels/panel-01/
https://www.the-scientist.com/....daily-news/hiv-sprea
The Story of Gaetan Dugas:
https://www.lrb.co.uk/the-pape....r/v40/n18/tom-crewe/
https://www.scribd.com/book/414171918
https://www.scribd.com/book/240406939
https://www.scribd.com/book/362833379
HIV/AIDS Today in Africa:
https://www.unaids.org/en/reso....urces/presscentre/pr
20AIDS%20mortality%20levels.
https://aids2020.unaids.org/ch....apter/region-profile
Margo W. never expected to develop bladder cancer, let alone 2 cancers at the same time. She had no symptoms, pain or discomfort when one day she noticed blood in her urine. At first, she was diagnosed with stage 1 bladder cancer. But then, she learned she had a second cancer, plasmacytoid, that was described as “sneaky, asymptomatic and 100% fatal and recurring.”
#cancerstories #cancerpatient #cancersurvivor #patientstories #thepatientstory #BladderCancer #blcsm #BladdersMatter #urology #plasmacytoid #self-advocacy #secondopinion
Watch Margo's Video Series:
Segment 1: https://my.mtr.cool/pllsubedsg
Segment 2: https://my.mtr.cool/dnyzzugrzi
In cancer treatment, the precise dosing of chemotherapy is extremely important; it can be a challenge for doctors to find the maximum effective dose that will not be catastrophically toxic. Many chemotherapy drugs are oxidized by cytochrome P450 enzymes before their inactivation or excretion. This means that for patients also using CBD, the same dose of chemotherapy may produce higher blood concentrations. If CBD inhibits the metabolism of chemotherapy drugs and dosage adjustments aren’t made, the chemotherapy agent could accumulate within the body to highly toxic levels.
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Esophageal cancer kills most who are diagnosed with it. Symptoms tend to be acid reflux and trouble swallowing, but, by that point, the cancer is usually considered advanced. https://www.abcactionnews.com/....news/in-depth/in-dep
